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Manzoor, Irtiqa, Rasool, Akhter, S.S, Jasvinder. (1404). Multimodal Diagnosis and Management of Canine Ascites: A Clinical Case Report. سامانه مدیریت نشریات علمی, 80(3), 807-812. doi: 10.32592/ARI.2025.80.3.807
Irtiqa Manzoor; Akhter Rasool; Jasvinder S.S. "Multimodal Diagnosis and Management of Canine Ascites: A Clinical Case Report". سامانه مدیریت نشریات علمی, 80, 3, 1404, 807-812. doi: 10.32592/ARI.2025.80.3.807
Manzoor, Irtiqa, Rasool, Akhter, S.S, Jasvinder. (1404). 'Multimodal Diagnosis and Management of Canine Ascites: A Clinical Case Report', سامانه مدیریت نشریات علمی, 80(3), pp. 807-812. doi: 10.32592/ARI.2025.80.3.807
Manzoor, Irtiqa, Rasool, Akhter, S.S, Jasvinder. Multimodal Diagnosis and Management of Canine Ascites: A Clinical Case Report. سامانه مدیریت نشریات علمی, 1404; 80(3): 807-812. doi: 10.32592/ARI.2025.80.3.807

Multimodal Diagnosis and Management of Canine Ascites: A Clinical Case Report

Archives of Razi Institute
مقاله 31، دوره 80، شماره 3، مرداد و شهریور 2025، صفحه 807-812    اصل مقاله (522.24 K)
نوع مقاله: Case Study
شناسه دیجیتال (DOI): 10.32592/ARI.2025.80.3.807
نویسندگان
Irtiqa Manzoor1؛ Akhter Rasool* 2؛ Jasvinder S.S3
1Division of Veterinary Medicine, Sher-e-Kashmir University of Agricultural Sciences and Technology of Kashmir, Jammu, India.
2Department of Veterinary Gynaecology and Obstetrics, Madras Veterinary College, Chennai, India.
3Division of Veterinary Clinical Complex, Sher-e-Kashmir University of Agricultural Sciences and Technology of Kashmir, Jammu, India.
چکیده
Ascites, the pathological accumulation of fluid within the peritoneal cavity, often results from an imbalance between fluid production and absorption. This case involved a seven-and-a-half-year-old neutered female Labrador retriever that presented with abdominal distension, inappetence, weakness, and lethargy. Clinical examination revealed respiratory distress, abdominal pain, pale mucous membranes, muffled heart sounds, and a positive fluid thrill upon abdominal palpation. Hematological evaluation showed normocytic normochromic anemia, neutrophilia, leukocytosis, and thrombocytopenia. Biochemical analysis revealed hypoalbuminemia, hypoglycemia, elevated liver enzymes (ALT, AST, ALP), and increased blood urea nitrogen (BUN) and creatinine levels, pointing to impaired liver and kidney function. Arterial blood gas analysis indicated hepatobiliary dysfunction, showing low pCO₂, reduced bicarbonate (HCO₃) levels, and a negative base excess (BE), suggestive of metabolic acidosis with compensatory respiratory alkalosis. Radiographic imaging showed a ground-glass appearance and pleural effusion, while ultrasonography confirmed the presence of free anechoic fluid in the peritoneal cavity, rounded liver margins, a dilated hepatic portal vein, hyperechoic liver parenchyma, ill-defined corticomedullary junctions in the kidneys, and splenomegaly. Cytological analysis of the straw-colored ascitic fluid showed fibrin strands and white blood cells, with a serum-ascites albumin gradient (SAAG) of 2.4 g/dL, indicating portal hypertension as the underlying cause. The final diagnosis was ascites of hepatic origin. Treatment included diuretics, antibiotics, fluid therapy, liver supplements, and dietary modifications, including salt restriction and the provision of high-quality protein. This case underscores the importance of a comprehensive diagnostic approach, incorporating clinical, hematological, biochemical, and imaging findings to ensure timely intervention and effective management of canine ascites.
کلیدواژه‌ها
Abdominocentesis؛ Imaging؛ liver؛ Portal hypertension؛ Therapeutics
مراجع
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